PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9717846-4	1998	Rather, induction of PR mRNA depends on the differentiation of granulosa cells in response to E and a physiological amount of FSH followed by exposure to agonists (elevated levels of LH, FSH, and forskolin) that markedly increase cAMP.	Colforsin	196-205	progesterone receptor	Rattus norvegicus	21-23	
9717846-5	1998	Induction of PR mRNA by forskolin is blocked by the A-kinase inhibitor H89 and cycloheximide but not by the E antagonist, ICI 164,384.	Colforsin	24-33	progesterone receptor	Rattus norvegicus	13-15	
9717846-7	1998	When distal and proximal PR promoter-reporter constructs that are responsive to E in other cell types were transiently transfected into differentiated granulosa cells, forskolin, but not E, induced activity.	Colforsin	168-177	progesterone receptor	Rattus norvegicus	25-27	
9717846-11	1998	The GC-rich region of the distal PR promoter bound Sp1 and Sp3 but not C/EBPalpha/beta, indicating that factors binding to ERE3 interact synergistically with Sp1/Sp3 to confer increased responsiveness of the distal promoter to forskolin.	Colforsin	227-236	progesterone receptor	Rattus norvegicus	33-35	
8299566-3	1994	Gonadotropins also effectively induce PR mRNA expression in primary cultures of rat granulosa cells, and this action can be mimicked by agents that elevate intracellular cAMP (forskolin or 8-bromo-cAMP).	Colforsin	176-185	progesterone receptor	Rattus norvegicus	38-40	
7588292-1	1995	We have previously shown that both pituitary gonadotropins and forskolin induce progesterone receptor (PR) messenger RNA expression at the level of transcription in granulosa cells of the rat ovary.	Colforsin	63-72	progesterone receptor	Rattus norvegicus	80-101	
7588292-1	1995	We have previously shown that both pituitary gonadotropins and forskolin induce progesterone receptor (PR) messenger RNA expression at the level of transcription in granulosa cells of the rat ovary.	Colforsin	63-72	progesterone receptor	Rattus norvegicus	103-105	
8299566-7	1994	However, when luciferase fusion genes containing a 1375-basepair rat PR gene promoter were transiently transfected into rat granulosa cells, luciferase activity could be stimulated several-fold by hCG or forskolin, but not by estrogen.	Colforsin	204-213	progesterone receptor	Rattus norvegicus	69-71	
8344215-2	1993	The present study was undertaken to determine the effects of various agonists (LH, FSH, forskolin, and GnRH) known to stimulate ovulation on their abilities to induce progesterone receptor (PR) mRNA and protein in rat preovulatory follicles and in cultured rat granulosa cells exhibiting a preovulatory phenotype.	Colforsin	88-97	progesterone receptor	Rattus norvegicus	167-188	
8344215-2	1993	The present study was undertaken to determine the effects of various agonists (LH, FSH, forskolin, and GnRH) known to stimulate ovulation on their abilities to induce progesterone receptor (PR) mRNA and protein in rat preovulatory follicles and in cultured rat granulosa cells exhibiting a preovulatory phenotype.	Colforsin	88-97	progesterone receptor	Rattus norvegicus	190-192