PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28356389-5	2017	Mechanistically, c-Fos decreases expression and activity of the nuclear receptor LXRalpha, leading to increased hepatic cholesterol and accumulation of toxic oxysterols and bile acids.	Oxysterols	158-168	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	17-22	
15191540-6	2004	Additionally, on Western and Northern analysis, oxysterol treatment increased two other AP-1 proteins, Jun-D and c-Fos, whereas Fra-2, Jun-B, and c-Jun were not changed.	Oxysterols	48-57	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	113-118	
18317936-8	2009	These results demonstrate that oxysterol-induced IL-8 secretion is a calcium-dependent phenomenon involving the MEK/ERK1/2 pathway leading to the activation of IL-8 gene via AP-1 (c-fos).	Oxysterols	31-40	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	174-178	
18317936-8	2009	These results demonstrate that oxysterol-induced IL-8 secretion is a calcium-dependent phenomenon involving the MEK/ERK1/2 pathway leading to the activation of IL-8 gene via AP-1 (c-fos).	Oxysterols	31-40	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	180-185