PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22313587-1	2012	Long-chain fatty acids have been shown to suppress appetite and reduce energy intake (EI) by stimulating the release of gastrointestinal hormones such as cholecystokinin (CCK).	long-chain fatty acids	0-22	cholecystokinin	Homo sapiens	154-169	
24357536-2	2014	Although long-chain fatty acids (FAs, >= C12) are well known to stimulate CCK secretion, the CCK-releasing activities of other aliphatic compounds, such as aldehydes (Alds) or alcohols (Alcs), have not been studied.	long-chain fatty acids	9-31	cholecystokinin	Homo sapiens	77-80	
22313587-1	2012	Long-chain fatty acids have been shown to suppress appetite and reduce energy intake (EI) by stimulating the release of gastrointestinal hormones such as cholecystokinin (CCK).	long-chain fatty acids	0-22	cholecystokinin	Homo sapiens	171-174	
19837920-9	2010	CONCLUSION: Generation of long-chain fatty acids through hydrolysis of fat is a critical step for fat-induced stimulation of GLP-1 in humans; the signal is mediated via CCK release and CCK-1 receptors.	long-chain fatty acids	26-48	cholecystokinin	Homo sapiens	169-172	
19837920-9	2010	CONCLUSION: Generation of long-chain fatty acids through hydrolysis of fat is a critical step for fat-induced stimulation of GLP-1 in humans; the signal is mediated via CCK release and CCK-1 receptors.	long-chain fatty acids	26-48	cholecystokinin	Homo sapiens	185-188