PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18077578-1	2007	We recently demonstrated that melatonin, N-acetylserotonin (NAS), and N-acetyldopamine (NAD) attenuate the synthesis of lipopolysaccharide (LPS)-stimulated tumor necrosis factor-alpha (TNF-alpha) and the generation of oxidant radicals.	N-acetylserotonin	41-58	tumor necrosis factor	Homo sapiens	156-183	
18077578-6	2007	Incubating THP-1-derived monocytes with rising concentrations of NAS, NAD, NMD, or 4-O-MD markedly decreased LPS-stimulated TNF-alpha production, which was dose dependent and on the order of 96%-98%.	N-acetylserotonin	65-68	tumor necrosis factor	Homo sapiens	124-133	
18077578-8	2007	Our results indicated that the inhibitory effect of NAS, NAD, NMD, or 4-O-MD on LPS-induced TNF-alpha production and FeCl(2)-stimulated lipid peroxidation is robust and dose dependent.	N-acetylserotonin	52-55	tumor necrosis factor	Homo sapiens	92-101	
18077578-1	2007	We recently demonstrated that melatonin, N-acetylserotonin (NAS), and N-acetyldopamine (NAD) attenuate the synthesis of lipopolysaccharide (LPS)-stimulated tumor necrosis factor-alpha (TNF-alpha) and the generation of oxidant radicals.	N-acetylserotonin	41-58	tumor necrosis factor	Homo sapiens	185-194	
18077578-1	2007	We recently demonstrated that melatonin, N-acetylserotonin (NAS), and N-acetyldopamine (NAD) attenuate the synthesis of lipopolysaccharide (LPS)-stimulated tumor necrosis factor-alpha (TNF-alpha) and the generation of oxidant radicals.	N-acetylserotonin	60-63	tumor necrosis factor	Homo sapiens	156-183	
18077578-1	2007	We recently demonstrated that melatonin, N-acetylserotonin (NAS), and N-acetyldopamine (NAD) attenuate the synthesis of lipopolysaccharide (LPS)-stimulated tumor necrosis factor-alpha (TNF-alpha) and the generation of oxidant radicals.	N-acetylserotonin	60-63	tumor necrosis factor	Homo sapiens	185-194	
16179544-2	2005	In the present study, we examined whether melatonin, NAS, and N-acetyldopamine (NAD) have a modulatory effect on tumor necrosis factor-alpha (TNF-alpha) synthesis and superoxide production.	N-acetylserotonin	53-56	tumor necrosis factor	Homo sapiens	113-140	
16179544-2	2005	In the present study, we examined whether melatonin, NAS, and N-acetyldopamine (NAD) have a modulatory effect on tumor necrosis factor-alpha (TNF-alpha) synthesis and superoxide production.	N-acetylserotonin	53-56	tumor necrosis factor	Homo sapiens	142-151	
16179544-6	2005	Incubation of THP-1-derived monocytes with increasing concentrations of melatonin, NAS, or NAD resulted in a marked decrease in LPS-stimulated TNF-alpha production, which was dose-dependent and on the order of 96-98%.	N-acetylserotonin	83-86	tumor necrosis factor	Homo sapiens	143-152	
16179544-11	2005	Our results indicate that the inhibitory effect of melatonin, NAS, or NAD on LPS-induced TNF-alpha production is robust and dose-dependent.	N-acetylserotonin	62-65	tumor necrosis factor	Homo sapiens	89-98