PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17395747-3	2007	We presently studied the molecular regulation of Chop expression in insulin-producing cells (INS-1E) in response to three pro-apoptotic and endoplasmic reticulum stress-inducing agents, namely the cytokines interleukin-1beta + interferon-gamma, the free fatty acid palmitate, and the sarcoendoplasmic reticulum pump Ca(2+) ATPase blocker cyclopiazonic acid (CPA).	cyclopiazonic acid	338-356	DNA-damage inducible transcript 3	Rattus norvegicus	49-53	
17395747-3	2007	We presently studied the molecular regulation of Chop expression in insulin-producing cells (INS-1E) in response to three pro-apoptotic and endoplasmic reticulum stress-inducing agents, namely the cytokines interleukin-1beta + interferon-gamma, the free fatty acid palmitate, and the sarcoendoplasmic reticulum pump Ca(2+) ATPase blocker cyclopiazonic acid (CPA).	cyclopiazonic acid	358-361	DNA-damage inducible transcript 3	Rattus norvegicus	49-53	
17395747-4	2007	Detailed mutagenesis studies of the Chop promoter showed differential regulation of Chop transcription by CPA, cytokines, and palmitate.	cyclopiazonic acid	106-109	DNA-damage inducible transcript 3	Rattus norvegicus	36-40	
17395747-4	2007	Detailed mutagenesis studies of the Chop promoter showed differential regulation of Chop transcription by CPA, cytokines, and palmitate.	cyclopiazonic acid	106-109	DNA-damage inducible transcript 3	Rattus norvegicus	84-88	
16604358-11	2006	Furthermore, CPA-induced Chop mRNA expression and beta cell apoptosis were potentiated in cells that had been pretreated with IFN-gamma.	cyclopiazonic acid	13-16	DNA-damage inducible transcript 3	Rattus norvegicus	25-29