PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 30462625-0 2018 miR-339-5p Increases Radiosensitivity of Lung Cancer Cells by Targeting Phosphatases of Regenerating Liver-1 (PRL-1). mir-339-5p 0-10 protein tyrosine phosphatase 4A1 Homo sapiens 110-115 30462625-7 2018 Further exploration validated that miR-339-5p can target phosphatases of regenerating liver-1 (PRL-1) in lung cancer cells. mir-339-5p 35-45 protein tyrosine phosphatase 4A1 Homo sapiens 96-101 30462625-8 2018 Restoration of PRL-1 partially reverses the enhanced radiosensitivity of lung cancer cells induced by miR-339-5p. mir-339-5p 102-112 protein tyrosine phosphatase 4A1 Homo sapiens 15-20 26722251-3 2015 Previously, the present authors reported that miR-339-5p could inhibit cell proliferation and migration by targeting the 3"-untranslated region (3"-UTR) of PRL-1 mRNA. mir-339-5p 46-56 protein tyrosine phosphatase 4A1 Homo sapiens 156-161 23696794-0 2013 MiR-339-5p regulates the growth, colony formation and metastasis of colorectal cancer cells by targeting PRL-1. mir-339-5p 0-10 protein tyrosine phosphatase 4A1 Homo sapiens 105-110 23696794-3 2013 Here, we report that miR-339-5p is a tumor suppressor by regulating expression of PRL-1. mir-339-5p 21-31 protein tyrosine phosphatase 4A1 Homo sapiens 82-87 23696794-7 2013 Overexpression of miR-339-5p would also reduce the expression of PRL-1 mRNA and protein. mir-339-5p 18-28 protein tyrosine phosphatase 4A1 Homo sapiens 65-70 23696794-10 2013 In conclusion, our results demonstrate that miR-339-5p functions as a tumor suppressor and plays a role in inhibiting growth and metastasis of CRC cells through targeting PRL-1 and regulating p-ERK1/2 .These findings suggest that miR-339-5p may be useful as a new potential therapeutic target for CRC. mir-339-5p 44-54 protein tyrosine phosphatase 4A1 Homo sapiens 171-176 23696794-10 2013 In conclusion, our results demonstrate that miR-339-5p functions as a tumor suppressor and plays a role in inhibiting growth and metastasis of CRC cells through targeting PRL-1 and regulating p-ERK1/2 .These findings suggest that miR-339-5p may be useful as a new potential therapeutic target for CRC. mir-339-5p 230-240 protein tyrosine phosphatase 4A1 Homo sapiens 171-176