PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33962228-0	2021	LncRNA NEAT1 inhibition upregulates miR-16-5p to restrain the progression of sepsis-induced lung injury via suppressing BRD4 in a mouse model.	mir-16-5p	36-45	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	7-12	
33962228-2	2021	However, the role of nuclear paraspeckle assembly transcript 1 (NEAT1) binding miR-16-5p in sepsis-induced lung injury remains largely unknown.	mir-16-5p	79-88	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	64-69	
33962228-3	2021	We aim to explore the effect of NEAT1 sponging miR-16-5p on sepsis-induced lung injury via regulating bromodomain containing 4 (BRD4).	mir-16-5p	47-56	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	32-37	
33962228-10	2021	NEAT1 bound with miR-16-5p and miR-16-5p targeted BRD4.	mir-16-5p	17-26	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	0-5	
33962228-10	2021	NEAT1 bound with miR-16-5p and miR-16-5p targeted BRD4.	mir-16-5p	31-40	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	0-5	
33962228-11	2021	CONCLUSION: NEAT1 inhibition upregulates miR-16-5p to repress the progression of sepsis-induced lung injury via downregulating BRD4.	mir-16-5p	41-50	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	12-17	
32307795-0	2020	LncRNA NEAT1 promotes proliferation of chondrocytes via down-regulation of miR-16-5p in osteoarthritis.	mir-16-5p	75-84	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	7-12	
32307795-11	2020	CONCLUSION: The suppression of miR-16-5p, mediated by NEAT1 overexpression, could promote proliferation and inhibit apoptosis of chondrocytes.	mir-16-5p	31-40	nuclear paraspeckle assembly transcript 1 (non-protein coding)	Mus musculus	54-59