PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34039959-4	2021	Mechanistically, BKM120 decreased expression of HR molecules, including RAD51 and BRCA1/2, and reduced HR repair efficiency in GBM cells, therefore increasing levels of apoptosis induced by rucaparib.	NVP-BKM120	17-23	RAD51 recombinase	Homo sapiens	72-77	
33637776-11	2021	Meanwhile, BKM120 inhibited the expression of BRAC1/2 and Rad51/52 to block HR mediated repair through the PI3K/Akt/NFkappaB/c-Myc signaling pathway and PI3K/Akt/ FOXM1/Exo1 signaling pathway.	NVP-BKM120	11-17	RAD51 recombinase	Homo sapiens	58-63	
28945226-6	2018	Mechanistically, PI3K blockade by BKM120 attenuated HR competency with gammaH2AX accumulation and reduced RAD51 and BRCA1 expression in Ishikawa, AN3CA and Nou-1 cells, but the same combination treatment led to enhanced phosphorylation of DNA-PK, a non-homologous end joining repair protein, in Hec-108 cells.	NVP-BKM120	34-40	RAD51 recombinase	Homo sapiens	106-111