PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34377256-0	2021	Effect of miR-132-3p on sepsis-induced acute kidney injury in mice via regulating HAVCR1/KIM-1.	mir-132-3p	10-20	hepatitis A virus cellular receptor 1	Mus musculus	82-88	
34377256-0	2021	Effect of miR-132-3p on sepsis-induced acute kidney injury in mice via regulating HAVCR1/KIM-1.	mir-132-3p	10-20	hepatitis A virus cellular receptor 1	Mus musculus	89-94	
34377256-3	2021	Dual-luciferase reporter assay was performed to verify the targeting relationship between miR-132-3p and HAVCR1/KIM-1.	mir-132-3p	90-100	hepatitis A virus cellular receptor 1	Mus musculus	105-111	
34377256-3	2021	Dual-luciferase reporter assay was performed to verify the targeting relationship between miR-132-3p and HAVCR1/KIM-1.	mir-132-3p	90-100	hepatitis A virus cellular receptor 1	Mus musculus	112-117	
34377256-8	2021	MiR-132-3p overexpression could improve the renal function of the mice with sepsis and inhibit renal cell apoptosis and inflammatory progression, whereas HAVCR1/KIM1 overexpression exhibited an opposite effect and could block the renal protective effects of miR-132-3p overexpression on the septic mice.	mir-132-3p	258-268	hepatitis A virus cellular receptor 1	Mus musculus	154-160	
34377256-8	2021	MiR-132-3p overexpression could improve the renal function of the mice with sepsis and inhibit renal cell apoptosis and inflammatory progression, whereas HAVCR1/KIM1 overexpression exhibited an opposite effect and could block the renal protective effects of miR-132-3p overexpression on the septic mice.	mir-132-3p	258-268	hepatitis A virus cellular receptor 1	Mus musculus	161-165	
34377256-9	2021	CONCLUSION: MiR-132-3p overexpression can inhibit renal cell apoptosis and inflammatory progression via suppressing HAVCR1/KIM-1 expression, thereby exert renal protective effects on mice with sepsis.	mir-132-3p	12-22	hepatitis A virus cellular receptor 1	Mus musculus	116-122	
34377256-9	2021	CONCLUSION: MiR-132-3p overexpression can inhibit renal cell apoptosis and inflammatory progression via suppressing HAVCR1/KIM-1 expression, thereby exert renal protective effects on mice with sepsis.	mir-132-3p	12-22	hepatitis A virus cellular receptor 1	Mus musculus	123-128