PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19909294-9	2010	PEA (5-10 mg/kg, intraperitoneal) also inhibited DNFB-induced ear inflammation in mice in vivo, in a way attenuated by TRPV1 antagonism.	palmidrol	0-3	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	119-124	
25205418-1	2015	BACKGROUND AND PURPOSE: Palmitoylethanolamide (PEA) acts via several targets, including cannabinoid CB1 and CB2 receptors, transient receptor potential vanilloid type-1 (TRPV1) ion channels, peroxisome proliferator-activated receptor alpha (PPAR alpha) and orphan G protein-coupled receptor 55 (GRR55), all involved in the control of intestinal inflammation.	palmidrol	24-45	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	123-168	
25205418-1	2015	BACKGROUND AND PURPOSE: Palmitoylethanolamide (PEA) acts via several targets, including cannabinoid CB1 and CB2 receptors, transient receptor potential vanilloid type-1 (TRPV1) ion channels, peroxisome proliferator-activated receptor alpha (PPAR alpha) and orphan G protein-coupled receptor 55 (GRR55), all involved in the control of intestinal inflammation.	palmidrol	24-45	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	170-175	
25205418-1	2015	BACKGROUND AND PURPOSE: Palmitoylethanolamide (PEA) acts via several targets, including cannabinoid CB1 and CB2 receptors, transient receptor potential vanilloid type-1 (TRPV1) ion channels, peroxisome proliferator-activated receptor alpha (PPAR alpha) and orphan G protein-coupled receptor 55 (GRR55), all involved in the control of intestinal inflammation.	palmidrol	47-50	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	123-168	
24818658-1	2014	BACKGROUND AND PURPOSE: Palmitoylethanolamide (PEA), a naturally occurring acylethanolamide chemically related to the endocannabinoid anandamide, interacts with targets that have been identified in peripheral nerves controlling gastrointestinal motility, such as cannabinoid CB1 and CB2 receptors, TRPV1 channels and PPARalpha.	palmidrol	47-50	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	298-303	
25205418-1	2015	BACKGROUND AND PURPOSE: Palmitoylethanolamide (PEA) acts via several targets, including cannabinoid CB1 and CB2 receptors, transient receptor potential vanilloid type-1 (TRPV1) ion channels, peroxisome proliferator-activated receptor alpha (PPAR alpha) and orphan G protein-coupled receptor 55 (GRR55), all involved in the control of intestinal inflammation.	palmidrol	47-50	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	170-175	
24818658-0	2014	Palmitoylethanolamide normalizes intestinal motility in a model of post-inflammatory accelerated transit: involvement of CB1 receptors and TRPV1 channels.	palmidrol	0-21	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	139-144	
24818658-1	2014	BACKGROUND AND PURPOSE: Palmitoylethanolamide (PEA), a naturally occurring acylethanolamide chemically related to the endocannabinoid anandamide, interacts with targets that have been identified in peripheral nerves controlling gastrointestinal motility, such as cannabinoid CB1 and CB2 receptors, TRPV1 channels and PPARalpha.	palmidrol	24-45	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	298-303	
18602217-0	2008	The endogenous fatty acid amide, palmitoylethanolamide, has anti-allodynic and anti-hyperalgesic effects in a murine model of neuropathic pain: involvement of CB(1), TRPV1 and PPARgamma receptors and neurotrophic factors.	palmidrol	33-54	transient receptor potential cation channel, subfamily V, member 1	Mus musculus	166-171