PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30485521-12	2018	MiR-181a can inhibit the apoptosis of hypoxic-reoxygenated cardiomyocytes by targeting XIAP to downregulate Bax and upregulate Bcl expression.	mir-181a	0-8	BCL2 associated X, apoptosis regulator	Homo sapiens	108-111	
29907248-7	2018	Overexpression of miR-181a strongly reduced the cell viability of MCF-7 cells, mainly through increased apoptosis, which was evidenced by substantially increased gene expression of pro-apoptosis factors such as BAX and CASPASE 9 in miR-181a overexpressed cells.	mir-181a	18-26	BCL2 associated X, apoptosis regulator	Homo sapiens	211-214	
27534652-10	2016	In addition, inhibition of miR-181a promoted apoptosis of HeLa and CaSKi cells due to increasing Bax expression and decreasing Bcl-2 expression.	mir-181a	27-35	BCL2 associated X, apoptosis regulator	Homo sapiens	97-100