PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32431516-12	2020	Silencing of STC1 reverses the tumor promoting effects of MALAT1 up-regulation and miR-101-3p down-regulation on CC cells.	mir-101-3p	83-93	stanniocalcin 1	Homo sapiens	13-17	
34650661-11	2021	Furthermore, stanniocalcin-1 (STC1) was selected to be the target gene of miR-101-3p according to the bioinformatic algorithms.	mir-101-3p	74-84	stanniocalcin 1	Homo sapiens	13-28	
34650661-11	2021	Furthermore, stanniocalcin-1 (STC1) was selected to be the target gene of miR-101-3p according to the bioinformatic algorithms.	mir-101-3p	74-84	stanniocalcin 1	Homo sapiens	30-34	
34650661-12	2021	Mechanically, upregulation of miR-101-3p significantly decreased the expression of STC1, vascular endothelial growth factor (VEGF), and MAPK pathway expression levels.	mir-101-3p	30-40	stanniocalcin 1	Homo sapiens	83-87	
34650661-15	2021	Our findings demonstrated that miR-101-3p alleviated IDD process through the STC1/VEGF/MAPK pathway.	mir-101-3p	31-41	stanniocalcin 1	Homo sapiens	77-81	
33784880-16	2021	CONCLUSION: It could be concluded that HOTTIP sponged miR-101-3p to upregulate STC1 expression, thereby promoting RB cell proliferation and inhibiting apoptosis.	mir-101-3p	54-64	stanniocalcin 1	Homo sapiens	79-83