PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29877111-0	2019	PEITC promotes neurite growth in primary sensory neurons via the miR-17-5p/STAT3/GAP-43 axis.	phenethyl isothiocyanate	0-5	microRNA 17	Homo sapiens	65-74	
29877111-6	2019	To further determine a substitution therapy for sciatic nerve conditioning injury, beta-phenethyl isothiocyanate (PEITC), which downregulates miR-17-5p, was assessed.	phenethyl isothiocyanate	83-112	microRNA 17	Homo sapiens	142-151	
29877111-6	2019	To further determine a substitution therapy for sciatic nerve conditioning injury, beta-phenethyl isothiocyanate (PEITC), which downregulates miR-17-5p, was assessed.	phenethyl isothiocyanate	114-119	microRNA 17	Homo sapiens	142-151	
29877111-8	2019	Further experiments demonstrated PEITC induced neurite growth by inhibiting miR-17-5p and further upregulating STAT3, p-STAT3 and GAP-43.	phenethyl isothiocyanate	33-38	microRNA 17	Homo sapiens	76-85	
29877111-11	2019	PEITC could promote repair of injured dorsal column via the miR-17-5p/STAT3/GAP-43 axis, and could mimic the treatment effect of sciatic nerve conditioning injury.	phenethyl isothiocyanate	0-5	microRNA 17	Homo sapiens	60-69	
23661605-8	2013	PEITC treatment significantly decreased PCAF expression and promoted transcription of miR-17 in LNCaP cells.	phenethyl isothiocyanate	0-5	microRNA 17	Homo sapiens	86-92	
24732804-2	2014	Here we demonstrate that PEITC-induced ROS decreased expression of microRNA 27a (miR-27a)/miR-20a:miR-17-5p and induced miR-regulated ZBTB10/ZBTB4 and ZBTB34 transcriptional repressors, which, in turn, downregulate specificity protein (Sp) transcription factors (TFs) Sp1, Sp3, and Sp4 in pancreatic cancer cells.	phenethyl isothiocyanate	25-30	microRNA 17	Homo sapiens	98-107	
24732804-3	2014	Decreased expression of miR-27a/miR-20a:miR-17-5p by PEITC-induced ROS is a key step in triggering the miR-ZBTB Sp cascade leading to downregulation of Sp TFs, and this is due to ROS-dependent epigenetic effects associated with genome-wide shifts in repressor complexes, resulting in decreased expression of Myc and the Myc-regulated miRs.	phenethyl isothiocyanate	53-58	microRNA 17	Homo sapiens	40-49	
23661605-9	2013	Functional inhibition of miR-17 attenuated the suppression of PCAF in cells treated by PEITC.	phenethyl isothiocyanate	87-92	microRNA 17	Homo sapiens	25-31	
23661605-10	2013	CONCLUSION: Our results indicate that PEITC inhibits AR-regulated transcriptional activity and cell growth of PCa cells through miR-17-mediated suppression of PCAF, suggesting a new mechanism by which PEITC modulates PCa cell growth.	phenethyl isothiocyanate	38-43	microRNA 17	Homo sapiens	128-134	
23661605-10	2013	CONCLUSION: Our results indicate that PEITC inhibits AR-regulated transcriptional activity and cell growth of PCa cells through miR-17-mediated suppression of PCAF, suggesting a new mechanism by which PEITC modulates PCa cell growth.	phenethyl isothiocyanate	201-206	microRNA 17	Homo sapiens	128-134