PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16002552-7	2005	Both Ro 32-0432 (a protein kinase C inhibitor) and H89 (a protein kinase A inhibitor) effectively suppressed the PrRP31 (100 nM)-induced TH mRNA level.	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	51-54	tyrosine hydroxylase	Rattus norvegicus	137-139	
25957836-7	2015	The increase of TH was also inhibited by SQ22536 (adenylyl cyclase inhibitor), H-89(PKA inhibitor) and LY294002 (phosphatidylinositol 3 kinase inhibitor).	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	79-83	tyrosine hydroxylase	Rattus norvegicus	16-18	
16452470-6	2006	In contrast, protein kinase A inhibitor H-89 or Ca(2+)/calmodulin-activated protein kinase inhibitor KN-93 reduced the nicotine-triggered rise in P-CREB and TH promoter activity.	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	40-44	tyrosine hydroxylase	Rattus norvegicus	157-159	
26225746-7	2015	Notably, this reduction in the total-TH protein level was sensitive not only to a 26S proteasomal inhibitor, MG-132, but also to a PKA inhibitor, H-89.	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	146-150	tyrosine hydroxylase	Rattus norvegicus	37-39