PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16845490-14	2006	In addiction, PKA inhibitor H89 also inhibits c-Fos induction in 3T3-AR7 cells by ACTH(39), implicating activation of the cAMP/PKA/CREB pathway in c-Fos induction by ACTH(39).	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	28-31	FBJ osteosarcoma oncogene	Mus musculus	46-51	
16845490-14	2006	In addiction, PKA inhibitor H89 also inhibits c-Fos induction in 3T3-AR7 cells by ACTH(39), implicating activation of the cAMP/PKA/CREB pathway in c-Fos induction by ACTH(39).	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	28-31	FBJ osteosarcoma oncogene	Mus musculus	147-152	
10614650-3	2000	The specific protein kinase A (PKA) inhibitor, H89, inhibited c-fos induction.	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	47-50	FBJ osteosarcoma oncogene	Mus musculus	62-67	
8078487-9	1994	The specific protein kinase A inhibitor H-89 blocked increases in c-fos levels and [3H]thymidine incorporation induced by low concentrations of bombesin.	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	40-44	FBJ osteosarcoma oncogene	Mus musculus	66-71	
10535756-0	1999	Inhibition of 12-O-tetradecanoylphorbol-13-acetate induction of c-fos mRNA by the protein kinase A inhibitor N-[2-(p-bromocinnamylamino)ethyl]-5-isoquinoline sulfonamide.	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	109-169	FBJ osteosarcoma oncogene	Mus musculus	64-69	
10535756-3	1999	Treatment of NIH 3T3 cells with N-[2-(p-bromocinnamylamino)ethyl]-5-isoquinoline sulfonamide (H-89), a PKA specific inhibitor, suppressed TPA induction of PKA activity and c-fos mRNA in a concentration-dependent manner, but did not inhibit serum-induced transcription.	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	32-92	FBJ osteosarcoma oncogene	Mus musculus	172-177	
10535756-3	1999	Treatment of NIH 3T3 cells with N-[2-(p-bromocinnamylamino)ethyl]-5-isoquinoline sulfonamide (H-89), a PKA specific inhibitor, suppressed TPA induction of PKA activity and c-fos mRNA in a concentration-dependent manner, but did not inhibit serum-induced transcription.	N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide	94-98	FBJ osteosarcoma oncogene	Mus musculus	172-177