PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34628148-6	2021	Meanwhile, BA pretreatment markedly attenuated T-2 toxin-induced renal inflammatory response by decreasing the mRNA expression of IL-1beta, TNF-alpha and IL-10, and increasing IL-6 mRNA expression.	betulinic acid	11-13	interleukin 1 alpha	Mus musculus	130-138	
33322178-9	2020	Furthermore, treatment with 1 mg/kg BA downregulated the expression of p-NF-kappaB and p-IkappaB-alpha proteins in the intestine, while all doses of BA suppressed the pro-inflammatory cytokines expression of IL-1beta, IL-6 and TNF-alpha mRNAs and increased the anti-inflammatory cytokine expression of IL-10 mRNA in the intestine of T-2 toxin-exposed mice.	betulinic acid	36-38	interleukin 1 alpha	Mus musculus	208-216	
31545273-5	2019	BA reduced the serum levels of pro-inflammatory cytokines, such as IL-1alpha, IL-1beta, IL-5, IL-6, GM-CSF, KC, MCP-1 and PGE2 in Carr-treated mice, and increased those of anti-inflammatory cytokines, such as IL-12.	betulinic acid	0-2	interleukin 1 alpha	Mus musculus	67-76