PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17541273-7	2007	RESULTS: Treatment of EoL-1 cells with apicidin at 100 nM or n-butyrate at 500 microM decreased the levels of FIP1L1-PDGFRalpha protein and phosphorylated-Stat5, while that with trichostatin A at 30 nM did not.	apicidin	39-47	factor interacting with PAPOLA and CPSF1	Homo sapiens	110-116	
18504399-3	2008	The HDAC inhibitors apicidin and n-butyrate suppress the proliferation of EoL-1 cells and induce differentiation into eosinophils by a decrease in the protein level of FIP1L1-PDGFRalpha without affecting the mRNA level for FIP1L1-PDGFRA.	apicidin	20-28	factor interacting with PAPOLA and CPSF1	Homo sapiens	168-174	
18504399-3	2008	The HDAC inhibitors apicidin and n-butyrate suppress the proliferation of EoL-1 cells and induce differentiation into eosinophils by a decrease in the protein level of FIP1L1-PDGFRalpha without affecting the mRNA level for FIP1L1-PDGFRA.	apicidin	20-28	factor interacting with PAPOLA and CPSF1	Homo sapiens	223-229	
18504399-4	2008	In this study, we analyzed the mechanism by which the protein level of FIP1L1-PDGFRalpha is decreased by apicidin and n-butyrate.	apicidin	105-113	factor interacting with PAPOLA and CPSF1	Homo sapiens	71-77	
18504399-8	2008	RESULTS: When apicidin- and n-butyrate-treated EoL-1 cells were incubated in the presence of actinomycin D, the decrease in the protein level of FIP1L1-PDGFRalpha was significantly enhanced when compared with controls.	apicidin	14-22	factor interacting with PAPOLA and CPSF1	Homo sapiens	145-151	
17541273-8	2007	CONCLUSIONS: The decrease in the level of FIP1L1-PDGFRalpha protein caused by apicidin and n-butyrate might be one of the mechanisms by which EoL-1 cells are induced to differentiate into eosinophils by these HDAC inhibitors.	apicidin	78-86	factor interacting with PAPOLA and CPSF1	Homo sapiens	42-48