PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16685407-10	2006	The mucosal mRNA levels of eNOS were decreased after DSS administration, but preserved in mice treated with rosuvastatin.	Rosuvastatin Calcium	108-120	nitric oxide synthase 3, endothelial cell	Mus musculus	27-31	
16685407-11	2006	These results suggest that rosuvastatin prevents the development of DSS-induced colitis in mice via the inhibition of mucosal inflammatory responses associated with the preservation of eNOS transcription.	Rosuvastatin Calcium	27-39	nitric oxide synthase 3, endothelial cell	Mus musculus	185-189	
15563531-10	2005	Although we were unable to demonstrate that the myocardial protection was due to an effect on neutrophil infiltration, stem cell mobilization, or induction of NOS3, these data suggest that rosuvastatin may be particularly beneficial in myocardial protection after ischemia-reperfusion injury.	Rosuvastatin Calcium	189-201	nitric oxide synthase 3, endothelial cell	Mus musculus	159-163	
15625301-8	2005	Cotreatment with rosuvastatin prevented the decrease in eNOS, reduced ERK-1/-2 and normalized p38 levels.	Rosuvastatin Calcium	17-29	nitric oxide synthase 3, endothelial cell	Mus musculus	56-60	
12354446-7	2002	In addition, rosuvastatin increased myocardial endothelial nitric oxide synthase (eNOS) messenger ribonucleic acid levels.	Rosuvastatin Calcium	13-25	nitric oxide synthase 3, endothelial cell	Mus musculus	47-80	
18162625-12	2008	rosuvastatin was achieved with peak plasma concentrations <0.5 ng ml(-1) (ie, with 0.2 mg kg(-1)) and was associated with increased levels of phosphorylated Akt kinase and endothelial nitric oxide synthase in the vasculature.	Rosuvastatin Calcium	0-12	nitric oxide synthase 3, endothelial cell	Mus musculus	175-208	
23704894-0	2013	Rosuvastatin enhances angiogenesis via eNOS-dependent mobilization of endothelial progenitor cells.	Rosuvastatin Calcium	0-12	nitric oxide synthase 3, endothelial cell	Mus musculus	39-43	
23704894-8	2013	Interestingly, the enhanced circEPCs and post-HLI neovascularization stimulated by Ros were blunted in mice deficient in endothelial nitric oxide synthase (eNOS), and Ros increased p-Akt/p-eNOS levels in EPCs in vitro, indicating these effects of Ros are dependent on eNOS activity.	Rosuvastatin Calcium	83-86	nitric oxide synthase 3, endothelial cell	Mus musculus	121-154	
23704894-8	2013	Interestingly, the enhanced circEPCs and post-HLI neovascularization stimulated by Ros were blunted in mice deficient in endothelial nitric oxide synthase (eNOS), and Ros increased p-Akt/p-eNOS levels in EPCs in vitro, indicating these effects of Ros are dependent on eNOS activity.	Rosuvastatin Calcium	83-86	nitric oxide synthase 3, endothelial cell	Mus musculus	156-160	
23704894-8	2013	Interestingly, the enhanced circEPCs and post-HLI neovascularization stimulated by Ros were blunted in mice deficient in endothelial nitric oxide synthase (eNOS), and Ros increased p-Akt/p-eNOS levels in EPCs in vitro, indicating these effects of Ros are dependent on eNOS activity.	Rosuvastatin Calcium	83-86	nitric oxide synthase 3, endothelial cell	Mus musculus	189-193	
23704894-8	2013	Interestingly, the enhanced circEPCs and post-HLI neovascularization stimulated by Ros were blunted in mice deficient in endothelial nitric oxide synthase (eNOS), and Ros increased p-Akt/p-eNOS levels in EPCs in vitro, indicating these effects of Ros are dependent on eNOS activity.	Rosuvastatin Calcium	83-86	nitric oxide synthase 3, endothelial cell	Mus musculus	189-193	
23704894-8	2013	Interestingly, the enhanced circEPCs and post-HLI neovascularization stimulated by Ros were blunted in mice deficient in endothelial nitric oxide synthase (eNOS), and Ros increased p-Akt/p-eNOS levels in EPCs in vitro, indicating these effects of Ros are dependent on eNOS activity.	Rosuvastatin Calcium	167-170	nitric oxide synthase 3, endothelial cell	Mus musculus	189-193	
23704894-8	2013	Interestingly, the enhanced circEPCs and post-HLI neovascularization stimulated by Ros were blunted in mice deficient in endothelial nitric oxide synthase (eNOS), and Ros increased p-Akt/p-eNOS levels in EPCs in vitro, indicating these effects of Ros are dependent on eNOS activity.	Rosuvastatin Calcium	167-170	nitric oxide synthase 3, endothelial cell	Mus musculus	189-193	
23704894-8	2013	Interestingly, the enhanced circEPCs and post-HLI neovascularization stimulated by Ros were blunted in mice deficient in endothelial nitric oxide synthase (eNOS), and Ros increased p-Akt/p-eNOS levels in EPCs in vitro, indicating these effects of Ros are dependent on eNOS activity.	Rosuvastatin Calcium	167-170	nitric oxide synthase 3, endothelial cell	Mus musculus	189-193	
23704894-8	2013	Interestingly, the enhanced circEPCs and post-HLI neovascularization stimulated by Ros were blunted in mice deficient in endothelial nitric oxide synthase (eNOS), and Ros increased p-Akt/p-eNOS levels in EPCs in vitro, indicating these effects of Ros are dependent on eNOS activity.	Rosuvastatin Calcium	167-170	nitric oxide synthase 3, endothelial cell	Mus musculus	189-193	
23704894-9	2013	We conclude that Ros increases circEPCs and promotes their de novo differentiation through eNOS pathway.	Rosuvastatin Calcium	17-20	nitric oxide synthase 3, endothelial cell	Mus musculus	91-95