PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34352791-19	2021	Finally, we demonstrated silencing XIST significantly recovered miR-101-3p expression and downregulated expression of glycolysis key enzymes, a phenotype could be further overridden by miR-101-3p inhibition.	mir-101	64-71	X inactive specific transcript	Homo sapiens	35-39	
34352791-19	2021	Finally, we demonstrated silencing XIST significantly recovered miR-101-3p expression and downregulated expression of glycolysis key enzymes, a phenotype could be further overridden by miR-101-3p inhibition.	mir-101	185-192	X inactive specific transcript	Homo sapiens	35-39	
30472203-0	2019	LncRNA XIST promotes the epithelial to mesenchymal transition of retinoblastoma via sponging miR-101.	mir-101	93-100	X inactive specific transcript	Homo sapiens	7-11	
33057875-7	2021	Starbase bioinformatics prediction and luciferase assay were used to validate the relationship of miR-101-3p and XIST or CLDN1.	mir-101	98-105	X inactive specific transcript	Homo sapiens	113-117	
33057875-10	2021	XIST could directly bind with miR-101-3p.	mir-101	30-37	X inactive specific transcript	Homo sapiens	0-4	
33510942-5	2021	Furthermore, miR-101 could combine with both Xist and C/EBPalpha and KLF6 through the same microRNA response element (MRE) predicted by bioinformatics and verified by luciferase reporter assays.	mir-101	13-20	X inactive specific transcript	Homo sapiens	45-49	
33510942-6	2021	Moreover, we found that miR-101 knockdown restored the decreased M1 marker and the increased M2 marker expression and also reversed the promotion of proliferation and migration of human breast cancer cells (MCF-7) and human ovarian cancer (OV) cells caused by silencing Xist.	mir-101	24-31	X inactive specific transcript	Homo sapiens	270-274	
33510942-7	2021	Generally, the present study indicates that Xist could mediate macrophage polarization to affect cell proliferation and migration of breast and ovarian cancer by competing with miR-101 to regulate C/EBPalpha and KLF6 expression.	mir-101	177-184	X inactive specific transcript	Homo sapiens	44-48	
30472203-6	2019	Furthermore, the mechanism of XIST was mainly focused on miR-101/ZEB1 or ZEB2 signaling.	mir-101	57-64	X inactive specific transcript	Homo sapiens	30-34	
30472203-9	2019	Moreover, we found that XIST functioned as a competing endogenous RNA (ceRNA) for miR-101 to regulate the de-repression of its endogenous targets ZEB1 and ZEB2.	mir-101	82-89	X inactive specific transcript	Homo sapiens	24-28	
30472203-10	2019	In conclusion, these findings suggest that XIST may facilitate the progression of RB through acting as a ceRNA for miR-101 to mediate the expression of ZEB1 and ZEB2.	mir-101	115-122	X inactive specific transcript	Homo sapiens	43-47	
29100288-0	2017	Long noncoding RNA XIST promotes malignancies of esophageal squamous cell carcinoma via regulation of miR-101/EZH2.	mir-101	102-109	X inactive specific transcript	Homo sapiens	19-23	
29100288-6	2017	Knockdown of XIST resulted in elevated expression of miR-101 and decreased expression of EZH2.	mir-101	53-60	X inactive specific transcript	Homo sapiens	13-17	
29100288-7	2017	Further analysis showed that XIST functioned as the competitive endogenous RNA of miR-101 to regulate EZH2 expression.	mir-101	82-89	X inactive specific transcript	Homo sapiens	29-33	
27620004-0	2016	Long non-coding RNA XIST regulates gastric cancer progression by acting as a molecular sponge of miR-101 to modulate EZH2 expression.	mir-101	97-104	X inactive specific transcript	Homo sapiens	20-24	
27620004-5	2016	The regulating relationship between lncRNA XIST and miR-101 was investigated in gastric cancer cells.	mir-101	52-59	X inactive specific transcript	Homo sapiens	43-47	
27620004-9	2016	Furthermore, an inverse relationship between lncRNA XIST and miR-101 was found.	mir-101	61-68	X inactive specific transcript	Homo sapiens	52-56	
27620004-10	2016	Polycomb group protein enhancer of zeste homolog 2 (EZH2), a direct target of miR-101, could mediated the biological effects that lncRNA XIST exerted.	mir-101	78-85	X inactive specific transcript	Homo sapiens	137-141