PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31697926-12	2020	Over expression of TLR4/MyD88/NFkappaB axis and its downstream pro-inflammatory mediators TNF-alpha, IL6 and IFN-gamma were observed in CCl4 induced mice, and these indices were abrogated significantly after GA treatment.	ginkgolide A	208-210	interleukin 6	Mus musculus	101-104	
35367419-8	2022	The regulatory role of GA in downregulating Col I, Col III, fibronectin in NRCFs, and enhancing levels of ANP, BNP and beta-MHC in NRCMs were reversed by MMP9 overexpression, so as the downregulation of IL-1beta, IL-6 and TNF-alpha in Ang II-induced NRCFs and NRCMs.	ginkgolide A	23-25	interleukin 6	Mus musculus	213-217	
33677255-4	2021	The increases of inflammatory factors including tumor necrosis factor (TNF)-alpha, interleukin (IL)-1beta and IL-6 in the kidneys of LPS-treated mice or NRK-52E cells were inhibited by GA administration.	ginkgolide A	185-187	interleukin 6	Mus musculus	110-114	
28394269-5	2017	We showed that GA could suppress the expression of pro-inflammatory mediators (cyclooxygenase-2 (COX-2) and nitric oxide (NO) and pro-inflammatory cytokines (tumor necrosis factor (TNF)-alpha, interleukin (IL)-6 and IL-1beta) in LPS-treated mouse peritoneal macrophages, mouse macrophage RAW264.7 cells, and differentiated human monocytes (dTHP-1) in vitro.	ginkgolide A	15-17	interleukin 6	Mus musculus	193-211	
28394269-7	2017	Consistently, GA was also shown to inhibit the LPS-stimulated release of TNF-alpha and IL-6 in mice.	ginkgolide A	14-16	interleukin 6	Mus musculus	87-91