PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29441927-0	2016	Ginkgolide A inhibits lipopolysaccharide-induced inflammatory response in human coronary artery endothelial cells via downregulation of TLR4-NF-kappaB signaling through PI3K/Akt pathway.	ginkgolide A	0-12	nuclear factor kappa B subunit 1	Homo sapiens	141-150	
29441927-8	2016	GA and CLI-095 abolished the LPS-induced inflammatory mediator release and NF-kappaB signaling activation, and GA reduced the TLR4 mRNA expression without affecting cell viability.	ginkgolide A	0-2	nuclear factor kappa B subunit 1	Homo sapiens	75-84	
29441927-10	2016	We conclude that GA could attenuate the LPS-induced inflammatory response in HCAECs and the anti-inflammatory activity might be associated with the inhibition of TLR4-NF-kappaB signaling through PI3K/AKT pathway.	ginkgolide A	17-19	nuclear factor kappa B subunit 1	Homo sapiens	167-176	
12619887-10	2003	EGb and its terpenoids (ginkgolide A, ginkgolide B and bilobalide) could antagonize the homocysteine effect on iNOS expression in macrophages via their antioxidant effect resulting in attenuation of NF-kappaB activation.	ginkgolide A	24-36	nuclear factor kappa B subunit 1	Homo sapiens	199-208