PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34550981-8	2021	Our study demonstrates that genetic variation in LTA4H, together with timing of asthma onset, may contribute to variability in montelukast response.	montelukast	127-138	leukotriene A4 hydrolase	Homo sapiens	49-54	
34550981-9	2021	European individuals with early-onset (<=18y) carrying at least one copy of rs2660845 have increased odd of exacerbation under montelukast treatment, presumably due to the up-regulation of LTA4H activity.	montelukast	127-138	leukotriene A4 hydrolase	Homo sapiens	189-194	
21385196-8	2012	The PEF and FEV(1 0) responses to montelukast in the A/A genotypes (n = 4) for the LTA4H SNP were significantly higher than those in the G allele carriers (A/G+G/G) (n = 17).	montelukast	34-45	leukotriene A4 hydrolase	Homo sapiens	83-88	
19214143-3	2009	We genotyped 26 SNPs that had previously been interrogated for association with montelukast response in five candidate genes (ABCC1, ALOX5, CYSLTR1, LTA4H, LTC4S) in a population of 577 asthmatics who participated in a clinical trial comparing intermittent and continuous-release zileuton to placebo.	montelukast	80-91	leukotriene A4 hydrolase	Homo sapiens	149-154