PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26282221-3	2015	This study investigates PFOS sonochemical degradation and its dependency on the initial concentration (10-460 muM), and the applied sound frequency (25 and 500 kHz, and 1 MHz).	perfluorooctane sulfonic acid	24-28	latexin	Homo sapiens	110-113	
26282221-4	2015	PFOS was degraded by sonochemical treatment at concentrations as high as 460 muM, as demonstrated by fluoride release and total organic content data.	perfluorooctane sulfonic acid	0-4	latexin	Homo sapiens	77-80	
24583810-4	2014	In the presence of ferric ion (100 muM), PFOS (20 muM) decreased to below the detection limit within 48 h, with the rate constant of 1.67 d(-1), which was 50 times higher than that by direct photolysis (0.033 d(-1)).	perfluorooctane sulfonic acid	41-45	latexin	Homo sapiens	35-38	
24583810-4	2014	In the presence of ferric ion (100 muM), PFOS (20 muM) decreased to below the detection limit within 48 h, with the rate constant of 1.67 d(-1), which was 50 times higher than that by direct photolysis (0.033 d(-1)).	perfluorooctane sulfonic acid	41-45	latexin	Homo sapiens	50-53	
21964418-4	2011	Although all of the oxidation reactions tested were more or less inhibited by PFOS, diclofenac 4"-hydroxylation mediated mainly by CYP2C9 was most strongly inhibited (K(i) value of 40 nM), followed by paclitaxel 6alpha-hydroxylation mediated mainly by CYP2C8 (K(i) value of 4 muM).	perfluorooctane sulfonic acid	78-82	latexin	Homo sapiens	276-279	
21964418-5	2011	The substrate oxidation reactions catalyzed by CYP2A6, CYP2B6, CYP2C19 and CYP3A4 were moderately (K(i) values of 35 to 45 muM), and those by CYP1A2, CYP2D6 and CYP2E1 were weakly inhibited by PFOS (K(i) values of 190-300 muM).	perfluorooctane sulfonic acid	193-197	latexin	Homo sapiens	123-126