PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34761352-7	2021	In addition, combined ACY-241 and erlotinib treatment induced autophagy and subsequently, cell death by reducing AKT-mTOR activity and increasing phospho-AMPK signaling.	Erlotinib Hydrochloride	34-43	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	154-158	
26119936-7	2016	Erlotinib treatment also induced AMPK activation despite loss of LKB1 expression, which was partially reduced by the application of a calcium/calmodulin-dependent protein kinase kinase 2 inhibitor (STO-609) or calcium chelator (BAPTA-AM).	Erlotinib Hydrochloride	0-9	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	33-37