PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23171857-11	2012	Further, sildenafil treatment attenuated glomerulosclerosis and tubulointerstitial fibrosis in DSH rats and counteracted the increased expression of ED-1, TGF-beta1, and Bax and the decreased expression of Bcl-2 in the kidneys of these rats.	Sildenafil Citrate	9-19	transforming growth factor, beta 1	Rattus norvegicus	155-164	
26343345-0	2015	Atorvastatin and sildenafil decrease vascular TGF-beta levels and MMP-2 activity and ameliorate arterial remodeling in a model of renovascular hypertension.	Sildenafil Citrate	17-27	transforming growth factor, beta 1	Rattus norvegicus	46-54	
26343345-3	2015	We hypothesized that atorvastatin and sildenafil alone or in association exert antiproliferative effects by down-regulating MMP-2 and TGF-beta, thus reducing the vascular hypertrophy induced by two kidney, one clip (2K1C) hypertension.	Sildenafil Citrate	38-48	transforming growth factor, beta 1	Rattus norvegicus	134-142	
26343345-15	2015	Atorvastatin and sildenafil was associated with decreased vascular TGF-beta levels and MMP-2 activity in renovascular hypertensive rats, thus ameliorating the vascular remodeling.	Sildenafil Citrate	17-27	transforming growth factor, beta 1	Rattus norvegicus	67-75	
24134744-3	2013	AIMS: The aims of this study are to determine whether activation of TGF-beta1 and its downstream pathways is responsible for the reduced efficacy of the PDE5is in diabetic ED via abnormalities in cavernosal structures and to investigate the synergistic effects of the TGF-beta1 antagonist P144 and sildenafil on erectile response.	Sildenafil Citrate	298-308	transforming growth factor, beta 1	Rattus norvegicus	68-77	
28535536-7	2017	Treatment with sildenafil had a modest effect on erectile response and an insignificant suppressive effect on CVOD, apoptosis, fibrosis, and the TGF-beta1 pathway.	Sildenafil Citrate	15-25	transforming growth factor, beta 1	Rattus norvegicus	145-154	
29218089-4	2017	Moreover, sildenafil dramatically decreased the expression of transforming growth factor-beta1 (TGF-beta1), vascular endothelial growth factor (VEGF), and alpha-smooth muscle actin (alpha-SMA) in the grafted aortas and increased the concentrations of cyclic guanosine monophosphate (cGMP) and endothelial nitric oxide synthase (eNOS) in serum.	Sildenafil Citrate	10-20	transforming growth factor, beta 1	Rattus norvegicus	62-94	
29218089-4	2017	Moreover, sildenafil dramatically decreased the expression of transforming growth factor-beta1 (TGF-beta1), vascular endothelial growth factor (VEGF), and alpha-smooth muscle actin (alpha-SMA) in the grafted aortas and increased the concentrations of cyclic guanosine monophosphate (cGMP) and endothelial nitric oxide synthase (eNOS) in serum.	Sildenafil Citrate	10-20	transforming growth factor, beta 1	Rattus norvegicus	96-105	
32671702-0	2020	Platelet-rich plasma and/or sildenafil topical applications accelerate and better repair wound healing in rats through regulation of proinflammatory cytokines and collagen/TGF-beta1 pathway.	Sildenafil Citrate	28-38	transforming growth factor, beta 1	Rattus norvegicus	172-181