PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29470986-11	2018	These results supported our conclusion that silencing of HOTAIR decreased drug resistance of NSCLC cells to Crizotinib through inhibition of autophagy via suppressing phosphorylation of ULK1.	Crizotinib	108-118	unc-51 like autophagy activating kinase 1	Homo sapiens	186-190	
34957997-0	2021	Curcumin increases crizotinib sensitivity through the inactivation of autophagy via epigenetic modulation of the miR-142-5p/Ulk1 axis in non-small cell lung cancer.	Crizotinib	19-29	unc-51 like autophagy activating kinase 1	Homo sapiens	124-128	
34957997-11	2021	Collectively, our findings demonstrate that curcumin sensitizes NSCLC cells to crizotinib by inactivating autophagy through the regulation of miR-142-5p and its target Ulk1.	Crizotinib	79-89	unc-51 like autophagy activating kinase 1	Homo sapiens	168-172