PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33177878-10	2020	Moreover, miR-142-3p mimic enhanced HCC cell proliferation, migration, invasion and cell cycle, while its effects were abolished by Bach-1 overexpression.	mir-142-3p	10-20	BTB domain and CNC homolog 1	Homo sapiens	132-138	
30480817-11	2019	Furthermore, replacement of miR-142-3p could inhibit the proliferation, invasion, and migration in breast cancer potentially by targeting of Bach-1 mRNA and subsequent inhibition of CXCR4, MMP9, and VEGFR protein expressions.	mir-142-3p	28-38	BTB domain and CNC homolog 1	Homo sapiens	141-147	
30480817-13	2019	CONCLUSION: For the first time, our results revealed that miR-142-3p could target Bach-1in breast cancer cells leading to the reduction of EMT-related proteins and reduced cell proliferation, invasion, and migration.	mir-142-3p	58-68	BTB domain and CNC homolog 1	Homo sapiens	82-88	
30480817-0	2019	miR-142-3p as tumor suppressor miRNA in the regulation of tumorigenicity, invasion and migration of human breast cancer by targeting Bach-1 expression.	mir-142-3p	0-10	BTB domain and CNC homolog 1	Homo sapiens	133-139	
30480817-6	2019	miR-142-3p targeting of Bach-1 expression in MCF-7 and MDA-MB-468 breast cancer cells was evaluated using bioinformatics, qRT-PCR and western blot analyses.	mir-142-3p	0-10	BTB domain and CNC homolog 1	Homo sapiens	24-30