PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 35150455-17 2022 Analysis of cytokines suggested a dominant pro-inflammatory T-cell response and osteodestructive function of IL-6 in PLs judging by Th17/Tfh cell activation, Tregs inhibition and increased RANKL/OPG ratio. tregs 158-163 interleukin 6 Homo sapiens 109-113 34940981-9 2021 However, as for Tregs, these cells produced more IL-10 than non-Treg CD4 T cells upon IL-6 stimulation, and these IL-10 led to the inhibition of CCR4 and CCR6. tregs 16-21 interleukin 6 Homo sapiens 86-90 35093485-9 2022 Exposure to specific antigens induced CD38+ B cells to produce IL-6, that converted Tregs to Th17 cells. tregs 84-89 interleukin 6 Homo sapiens 63-67 35228811-11 2022 Conclusion: TIGIT+Tregs levels are significantly reduced in ACS, accompanied by upregulated IL-6 and downregulated TGF-beta expression. tregs 18-23 interleukin 6 Homo sapiens 92-96 35562918-6 2022 Moreover, blockade of Interleukin (IL)-6 and Intercellular Adhesion Molecule (ICAM)-1 in cocultures significantly decreased the expansion of Tregs, suggesting an IL-6 and ICAM-1 dependent pathway. tregs 141-146 interleukin 6 Homo sapiens 22-40 35562918-6 2022 Moreover, blockade of Interleukin (IL)-6 and Intercellular Adhesion Molecule (ICAM)-1 in cocultures significantly decreased the expansion of Tregs, suggesting an IL-6 and ICAM-1 dependent pathway. tregs 141-146 interleukin 6 Homo sapiens 162-166 23374147-8 2013 CONCLUSIONS: The increase in the Th1 response in the TIVA-TCI group and the reduction in Tregs in the BAL group seem to balance the immunosuppressive effect induced by IL-6. tregs 89-94 interleukin 6 Homo sapiens 168-172 32345003-1 2020 The aim of this study was to explore the role of IL-6-miR-210 in the regulation of Tregs function and atrial fibrosis in atrial fibrillation (AF). tregs 83-88 interleukin 6 Homo sapiens 49-53 31863784-6 2020 Blocking of IL-6 secreted from IL-33-matDCs suppressed the conversion of Tregs to Th17 cells, indicating the greater propensity to convert stable Tregs to Th17 cells is due to IL-6 signaling. tregs 73-78 interleukin 6 Homo sapiens 12-16 31863784-6 2020 Blocking of IL-6 secreted from IL-33-matDCs suppressed the conversion of Tregs to Th17 cells, indicating the greater propensity to convert stable Tregs to Th17 cells is due to IL-6 signaling. tregs 146-151 interleukin 6 Homo sapiens 12-16