PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31732956-0	2020	miR-140-3p exhibits repressive functions on preosteoblast viability and differentiation by downregulating MCF2L in osteoporosis.	mir-140-3p	0-10	mcf.2 transforming sequence-like	Mus musculus	106-111	
31732956-8	2020	Luciferase reporter assay confirmed that MCF2L could be directly targeted by miR-140-3p.	mir-140-3p	77-87	mcf.2 transforming sequence-like	Mus musculus	41-46	
31732956-9	2020	Moreover, we identified that the expression of MCF2L was negatively regulated by miR-140-3p.	mir-140-3p	81-91	mcf.2 transforming sequence-like	Mus musculus	47-52	
31732956-10	2020	From rescue assays, we discovered that knockdown of MCF2L weakened the promoting influence of miR-140-3p ablation on MC3T3-E1 cell viability and differentiation, and receded the suppressing impact of miR-140-3p reduction on MC3T3-E1 cell apoptosis.	mir-140-3p	94-104	mcf.2 transforming sequence-like	Mus musculus	52-57	
31732956-10	2020	From rescue assays, we discovered that knockdown of MCF2L weakened the promoting influence of miR-140-3p ablation on MC3T3-E1 cell viability and differentiation, and receded the suppressing impact of miR-140-3p reduction on MC3T3-E1 cell apoptosis.	mir-140-3p	200-210	mcf.2 transforming sequence-like	Mus musculus	52-57	
31732956-11	2020	Above all, this research disclosed that miR-140-3p repressed preosteoblast viability and differentiation while promoted preosteoblast apoptosis via targeting MCF2L.	mir-140-3p	40-50	mcf.2 transforming sequence-like	Mus musculus	158-163