PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33121100-5	2020	In cultured human BSM cells (hBSMCs), transfection with a synthetic miR-140-3p inhibitor caused an increase in CD38 protein, indicating that its basal protein expression is regulated by endogenous miR-140-3p.	mir-140-3p	68-78	CD38 molecule	Homo sapiens	111-115	
33121100-5	2020	In cultured human BSM cells (hBSMCs), transfection with a synthetic miR-140-3p inhibitor caused an increase in CD38 protein, indicating that its basal protein expression is regulated by endogenous miR-140-3p.	mir-140-3p	197-207	CD38 molecule	Homo sapiens	111-115	
33121100-7	2020	Transfection of the hBSMCs with miR-140-3p mimic inhibited the CD38 protein upregulation induced by IL-13.	mir-140-3p	32-42	CD38 molecule	Homo sapiens	63-67	
33121100-9	2020	Taken together, the current findings suggest that the downregulation of miR-140-3p induced by IL-13 might cause an upregulation of CD38 protein in BSM cells of the disease, although functional and pathological roles of the upregulated CD38 are still unclear.	mir-140-3p	72-82	CD38 molecule	Homo sapiens	131-135	
33121100-9	2020	Taken together, the current findings suggest that the downregulation of miR-140-3p induced by IL-13 might cause an upregulation of CD38 protein in BSM cells of the disease, although functional and pathological roles of the upregulated CD38 are still unclear.	mir-140-3p	72-82	CD38 molecule	Homo sapiens	235-239