PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31061311-4	2019	Additional experiments show that UA inhibits cell migration and epithelial-mesenchymal transition (EMT), including E-cadherin, Vimentin, Integrin, Twist, and Zeb1 biomakers.	ursolic acid	33-35	vimentin	Homo sapiens	127-135	
31281396-9	2019	(3) UA significantly decreased the expressions of N-Cadherin, Vimentin, Snail, Twist p-Axl, p-IKKalpha/beta, and p-NF-kappaB in BGC-823 and MGC-803 cells.	ursolic acid	4-6	vimentin	Homo sapiens	62-70	
30643062-5	2018	However, after ursolic acid treatment, the migration and invasion ability were significantly reduced, and the expression of E-cadherin was increased while N-cadherin and vimentin levels were decreased.	ursolic acid	15-27	vimentin	Homo sapiens	170-178	
28090191-5	2016	Furthermore, ursolic acid treatment blocked epithelial and mesenchymal transition (EMT) molecules by activating E-cadherin as an epithelial marker and attenuating Vimentin, and Twist as mesenchymal molecules.	ursolic acid	13-25	vimentin	Homo sapiens	163-171	
23511428-5	2013	In addition, after treatment with UA, the A549 cells showed decreased expression of astrocyte-elevated gene-1 (AEG-1) accompanied by upregulation of E-cadherin and downregulation of N-cadherin and vimentin, which have been reported to characterize the epithelial-mesenchymal transition (EMT).	ursolic acid	34-36	vimentin	Homo sapiens	197-205	
23511428-6	2013	Further results also confirmed that the expression of vimentin was decreased by the siRNA technique to directly knock down AEG-1 expression, indicating that AEG-1 was involved in UA-mediated EMT inhibition.	ursolic acid	179-181	vimentin	Homo sapiens	54-62