PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
35438581-0	2022	Ursolic acid protects against anoxic injury in cardiac microvascular endothelial cells by regulating intercellular adhesion molecule-1 and toll-like receptor 4/MyD88/NF-kappaB pathway.	ursolic acid	0-12	MYD88 innate immune signal transduction adaptor	Homo sapiens	160-165	
35438581-10	2022	CONCLUSIONS: Our results insinuated that UA could alleviate H/R-induced injuries in CMECs by regulating ICAM1 and TLR4/MyD88/NF-kappaB pathway.	ursolic acid	41-43	MYD88 innate immune signal transduction adaptor	Homo sapiens	119-124	
31855318-0	2020	The miRNA-149-5p/MyD88 axis is responsible for ursolic acid-mediated attenuation of the stemness and chemoresistance of non-small cell lung cancer cells.	ursolic acid	47-59	MYD88 innate immune signal transduction adaptor	Homo sapiens	17-22	
31855318-4	2020	Mechanistic investigations revealed that UA inhibited the miR-149-5p/MyD88 signaling, which is responsible for UA-mediated effects on the stemness of A549-PR cells.	ursolic acid	41-43	MYD88 innate immune signal transduction adaptor	Homo sapiens	69-74	
31855318-4	2020	Mechanistic investigations revealed that UA inhibited the miR-149-5p/MyD88 signaling, which is responsible for UA-mediated effects on the stemness of A549-PR cells.	ursolic acid	111-113	MYD88 innate immune signal transduction adaptor	Homo sapiens	69-74	
31855318-6	2020	Therefore, these results suggest that UA could attenuate the stemness and chemoresistance of NSCLC cells through targeting miR-149-5p/MyD88 axis.	ursolic acid	38-40	MYD88 innate immune signal transduction adaptor	Homo sapiens	134-139