PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26567247-12	2017	Our data suggest that UA inhibits miR-21 expression and increases PTEN expression, which in turn inhibits Akt and mTOR and restores normal levels of autophagy.	ursolic acid	22-24	microRNA 21a	Mus musculus	34-40	
26567247-13	2017	Conclusions: Our data suggest that podocyte injury is associated with reduced levels of autophagy during exposure to high glucose conditions, UA attenuated podocyte injury via an increase in autophagy through miR-21 inhibition and PTEN expression, which inhibit the abnormal activation of the PI3K/Akt/mTOR pathway.	ursolic acid	142-144	microRNA 21a	Mus musculus	209-215	
25903305-0	2015	Downregulation of miR-21 is involved in direct actions of ursolic acid on the heart: implications for cardiac fibrosis and hypertrophy.	ursolic acid	58-70	microRNA 21a	Mus musculus	18-24	
25903305-9	2015	Downregulation of miR-21 and p-ERK/ERK were observed in myocardial fibroblasts treated with UA in a dose-dependent manner compared with the control group both in vitro and in vivo.	ursolic acid	92-94	microRNA 21a	Mus musculus	18-24	
25903305-10	2015	CONCLUSIONS: Our study demonstrates that UA can inhibit myocardial fibrosis both in vitro and in vivo, and the effects of UA on myocardial fibrosis may be due to the inhibition of miR-21/ERK signaling pathways.	ursolic acid	122-124	microRNA 21a	Mus musculus	180-186