PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7716769-5	1995	In contrast, there was a dramatic difference in the ability of VP-16 and PMC to protect GSH against AAPH-induced oxidation: while PMC inhibited AAPH-induced oxidation of GSH in a concentration-dependent manner, VP-16 did not protect GSH against oxidation.	2,2'-azobis(2-amidinopropane)	144-148	host cell factor C1	Homo sapiens	63-68	
7716769-3	1995	Using a water-soluble source of peroxyl radicals, the azo-initiator 2,2"-azobis(2-aminodinopropane) (AAPH), we found that both PMC and VP-16 are very efficient scavengers of peroxyl radicals as evidenced by their ability to inhibit AAPH-induced chemiluminescence of luminol and oxidation of PnA incorporated into DOPC liposomes.	2,2'-azobis(2-amidinopropane)	101-105	host cell factor C1	Homo sapiens	135-140	
7716769-3	1995	Using a water-soluble source of peroxyl radicals, the azo-initiator 2,2"-azobis(2-aminodinopropane) (AAPH), we found that both PMC and VP-16 are very efficient scavengers of peroxyl radicals as evidenced by their ability to inhibit AAPH-induced chemiluminescence of luminol and oxidation of PnA incorporated into DOPC liposomes.	2,2'-azobis(2-amidinopropane)	232-236	host cell factor C1	Homo sapiens	135-140	
7716769-5	1995	In contrast, there was a dramatic difference in the ability of VP-16 and PMC to protect GSH against AAPH-induced oxidation: while PMC inhibited AAPH-induced oxidation of GSH in a concentration-dependent manner, VP-16 did not protect GSH against oxidation.	2,2'-azobis(2-amidinopropane)	100-104	host cell factor C1	Homo sapiens	63-68	
7716769-5	1995	In contrast, there was a dramatic difference in the ability of VP-16 and PMC to protect GSH against AAPH-induced oxidation: while PMC inhibited AAPH-induced oxidation of GSH in a concentration-dependent manner, VP-16 did not protect GSH against oxidation.	2,2'-azobis(2-amidinopropane)	100-104	host cell factor C1	Homo sapiens	211-216	
7639514-4	1995	We found that VP-16 is an effective scavenger of peroxyl radicals as judged by its ability to inhibit a water-soluble azo-initiator, 2,2"-azobis(2-amidinopropane)dihydrochloride (AAPH)-induced (i) chemiluminescence (oxidation) of luminol, (ii) fluorescence decay (oxidation) of cis-parinaric acid incorporated in SR membranes, and (iii) peroxidation of SR membrane lipids.	2,2'-azobis(2-amidinopropane)	179-183	host cell factor C1	Homo sapiens	14-19	
7639514-6	1995	Electron spin resonance measurements showed that AAPH-induced VP-16 phenoxyl radicals were reduced by interaction with SR thiols.	2,2'-azobis(2-amidinopropane)	49-53	host cell factor C1	Homo sapiens	62-67