PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25034832-0	2014	Oxymatrine lightened the inflammatory response of LPS-induced mastitis in mice through affecting NF-kappaB and MAPKs signaling pathways.	oxymatrine	0-10	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	97-106	
25034832-6	2014	Oxymatrine inhibited the phosphorylation of NF-kappaB p65 and IkappaB in NF-kappaB signal pathway and reduced the phosphorylation of p38, ERK, and JNK in mitogen-activated protein kinase (MAPKs) signal pathway.	oxymatrine	0-10	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	44-53	
25034832-6	2014	Oxymatrine inhibited the phosphorylation of NF-kappaB p65 and IkappaB in NF-kappaB signal pathway and reduced the phosphorylation of p38, ERK, and JNK in mitogen-activated protein kinase (MAPKs) signal pathway.	oxymatrine	0-10	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	73-82	
25034832-7	2014	The results showed that oxymatrine had a protective effect on LPS-induced mastitis, and the anti-inflammatory mechanism of oxymatrine was related to the inhibition of NF-kappaB and MAPKs signal pathways.	oxymatrine	123-133	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	167-176	
24250585-0	2013	Anti-inflammatory Effects of Oxymatrine Through Inhibition of Nuclear Factor-kappa B and Mitogen-activated Protein Kinase Activation in Lipopolysaccharide-induced BV2 Microglia Cells.	oxymatrine	29-39	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	62-84	
24250585-3	2013	This study was designed to investigate the effects of oxymatrine on nuclear factor-kappa B (NF-kappaB) and mitogen-activated protein kinase (MAPK)-dependent inflammatory responses in lipopolysaccharide (LPS)-activated microglia.	oxymatrine	54-64	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	68-90	
24250585-3	2013	This study was designed to investigate the effects of oxymatrine on nuclear factor-kappa B (NF-kappaB) and mitogen-activated protein kinase (MAPK)-dependent inflammatory responses in lipopolysaccharide (LPS)-activated microglia.	oxymatrine	54-64	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	92-101	
32528295-0	2020	Oxymatrine Attenuates Dopaminergic Neuronal Damage and Microglia-Mediated Neuroinflammation Through Cathepsin D-Dependent HMGB1/TLR4/NF-kappaB Pathway in Parkinson"s Disease.	oxymatrine	0-10	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	133-142	
32528295-8	2020	It is worth noting that overexpression of CathD reversed OMT-targeted inhibition of HMGB1/TLR4/NF-kappaB signaling and OMT-produced neuroprotection in reconstituted neuron-microglia co-cultures.	oxymatrine	57-60	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	95-104	
27050799-7	2016	Furthermore, OMT administration inhibited the release of inflammatory factors including tumor necrosis factor-alpha, (TNF-alpha) interleukin-1beta (IL-1beta), and interleukin-6 (IL-6), as well as phosphorylated NF-kappaB p65.	oxymatrine	13-16	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	211-220