PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28927070-0	2017	Amentoflavone enhances sorafenib-induced apoptosis through extrinsic and intrinsic pathways in sorafenib-resistant hepatocellular carcinoma SK-Hep1 cells in vitro.	Sorafenib	23-32	DNL-type zinc finger	Homo sapiens	143-147	
34634308-11	2021	Interestingly, the combination of MDC and sorafenib significantly inhibited the invasion and metastasis of HCC cell lines SMMC-7721 and SK-Hep1 compared with the single drug treatment via the activated transcription factor FOXO1.	Sorafenib	42-51	DNL-type zinc finger	Homo sapiens	139-143	
28927070-0	2017	Amentoflavone enhances sorafenib-induced apoptosis through extrinsic and intrinsic pathways in sorafenib-resistant hepatocellular carcinoma SK-Hep1 cells in vitro.	Sorafenib	95-104	DNL-type zinc finger	Homo sapiens	143-147	
28927070-2	2017	The sorafenib-resistant SK-Hep1 (SK-Hep1R) cell line was established for the present study.	Sorafenib	4-13	DNL-type zinc finger	Homo sapiens	27-31	
28927070-3	2017	Initially, the differences in sorafenib-induced cytotoxicity and apoptosis between wild-type SK-Hep1 and SK-Hep1R cells were verified using the MTT assay and flow cytometry.	Sorafenib	30-39	DNL-type zinc finger	Homo sapiens	96-100	
28927070-5	2017	The results demonstrated that cell viability of SK-Hep1R cells was increased compared with that of SK-Hep1 cells following treatment with different concentrations of sorafenib for 24 h. Apoptosis of SK-Hep1R cells was lower than that of SK-Hep1 cells following treatment with 20 microM sorafenib for 24 h. Amentoflavone alone did not inhibit cell viability but significantly triggered sorafenib-induced cytotoxicity and apoptosis in SK-Hep1R cells.	Sorafenib	166-175	DNL-type zinc finger	Homo sapiens	51-55	
28927070-5	2017	The results demonstrated that cell viability of SK-Hep1R cells was increased compared with that of SK-Hep1 cells following treatment with different concentrations of sorafenib for 24 h. Apoptosis of SK-Hep1R cells was lower than that of SK-Hep1 cells following treatment with 20 microM sorafenib for 24 h. Amentoflavone alone did not inhibit cell viability but significantly triggered sorafenib-induced cytotoxicity and apoptosis in SK-Hep1R cells.	Sorafenib	166-175	DNL-type zinc finger	Homo sapiens	102-106	
23392173-2	2013	Sorafenib activated autophagy in a dose- and time-dependent manner in the HCC cell lines PLC5, Sk-Hep1, HepG2 and Hep3B.	Sorafenib	0-9	DNL-type zinc finger	Homo sapiens	98-102