PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32606928-0	2020	Hypoxia-Induced Aquaporin-3 Changes Hepatocellular Carcinoma Cell Sensitivity to Sorafenib by Activating the PI3K/Akt Signaling Pathway.	Sorafenib	81-90	aquaporin 3 (Gill blood group)	Homo sapiens	16-27	
34916846-0	2021	Hypoxia-Induced Aquaporin-3 Changes Hepatocellular Carcinoma Cell Sensitivity to Sorafenib by Activating the PI3K/Akt Signaling Pathway (Retraction).	Sorafenib	81-90	aquaporin 3 (Gill blood group)	Homo sapiens	16-27	
32606928-6	2020	Furthermore, the role of AQP3 in cell sensitivity to sorafenib was evaluated via flow cytometry, Western blotting, and a CCK-8 assay.	Sorafenib	53-62	aquaporin 3 (Gill blood group)	Homo sapiens	25-29	
32606928-9	2020	Compared with cells transfected with lentivirus-GFP (Lv-GFP), hypoxic cells transfected with lentivirus-AQP3 (Lv-AQP3) were less sensitive to sorafenib-induced apoptosis.	Sorafenib	142-151	aquaporin 3 (Gill blood group)	Homo sapiens	104-108	
32606928-9	2020	Compared with cells transfected with lentivirus-GFP (Lv-GFP), hypoxic cells transfected with lentivirus-AQP3 (Lv-AQP3) were less sensitive to sorafenib-induced apoptosis.	Sorafenib	142-151	aquaporin 3 (Gill blood group)	Homo sapiens	113-117	
32606928-14	2020	Conclusion: The study results show that AQP3 is a potential therapeutic target for improving the sensitivity of hypoxic HCC cells to sorafenib.	Sorafenib	133-142	aquaporin 3 (Gill blood group)	Homo sapiens	40-44