PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26489728-10	2015	Prasugrel treatment resulted in reduced MCP-1, ICAM-1 and TGF-beta mRNA levels on Day 2 (24h after the injury) and Day 8 (1 week after the injury) in the target arteries.	Prasugrel Hydrochloride	0-9	transforming growth factor, beta 1	Mus musculus	58-66	
26489728-11	2015	In conclusion, we found that a single oral loading dose of prasugrel markedly prevented neointimal hyperplasia by inhibiting platelet activation and thrombus formation and was associated with inhibition of the expression of inflammatory and fibrosis markers, including MCP-1, ICAM-1 and TGF-beta, in the injured arteries.	Prasugrel Hydrochloride	59-68	transforming growth factor, beta 1	Mus musculus	287-295