PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8592184-14	1995	The neuropeptide thyrotropin-releasing hormone (TRH), which also depolarizes and increases the RN of HMs, occluded the response of HMs to NE.	Metals, Heavy	101-104	thyrotropin releasing hormone	Rattus norvegicus	17-46	
8178024-2	1993	We found that TRH (0.1-10 microM) had two effects: 1) it caused depolarization of HMs and 2) decreased their input conductance.	Metals, Heavy	82-85	thyrotropin releasing hormone	Rattus norvegicus	14-17	
8178024-8	1993	This suggests that in the presence of TRH, the effects of both excitatory and inhibitory synaptic inputs on HMs are enhanced and the contrast between excitation and inhibition is sharpened.	Metals, Heavy	108-111	thyrotropin releasing hormone	Rattus norvegicus	38-41	
1479442-10	1992	Thus TRH acts directly on HMs to cause the depolarization.	Metals, Heavy	26-29	thyrotropin releasing hormone	Rattus norvegicus	5-8	
1479442-16	1992	Taken together, these results indicate that TRH depolarizes HMs, in part, by decreasing a resting K+ conductance.	Metals, Heavy	60-63	thyrotropin releasing hormone	Rattus norvegicus	44-47