PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20441599-7	2010	In contrast, Sal B and vitamin E significantly attenuated the expression of alpha-SMA, TGF-beta1, TbetaR-I, p-Smad2/3, and MMP-2 activity, but increased E-cadherin expression.	salvianolic acid B	13-18	matrix metallopeptidase 2	Rattus norvegicus	123-128	
20441599-9	2010	TGF-beta1 induced a typical EMT in HK-2 cells, while it was blocked by Sal B and SB-431542, as evidenced by blocking morphologic transformation, restoring E-cadherin and CK-18 expression, inhibiting alpha-SMA expression and F-actin reorganization, and down-regulating MMP-2/9 activities in TGF-beta1 mediated HK-2 cells.	salvianolic acid B	71-76	matrix metallopeptidase 2	Rattus norvegicus	268-273	
34636119-9	2022	The pharmacology network results showed that Sal B and its metabolites could regulate ALB, PLG, ACE, CASP3, MMP9, MMP2, MTOR, etc.	salvianolic acid B	45-50	matrix metallopeptidase 2	Rattus norvegicus	114-118	
30193865-10	2018	In the PPE-induced model, Sal-B reduced induction of lung"s matrix metalloproteinase (MMP)-9 and MMP-2 activities by 59 and 94%, respectively, and restored pSTAT3 and VEGF expressions to the healthy lung levels, while leaving neutrophil accumulation unchecked [myeloperoxidase (MPO) activity].	salvianolic acid B	26-31	matrix metallopeptidase 2	Rattus norvegicus	97-102