PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28852731-7	2016	RESULTS: Sal B significantly improved heart function and decreased infarct size; remarkably decreased levels of serum TNF-alpha and IL-Iotabeta levels, increased contents of myocardium antioxidant enzymes activities; western blot results showed that Sal B ameliorate the increased Bax and caspase-3 protins expressions and decreased Bcl-2 proteins expression and ratios of Bcl-2 to Bax.	salvianolic acid B	9-14	BCL2 associated X, apoptosis regulator	Rattus norvegicus	281-284	
35084700-8	2022	Furthermore, treatment with Sal B down-regulated HG-induced PARP, cleaved-caspase 3, cleaved-caspase 9, Bax, NLRP3, ASC, and IL-1 beta expression, but mitigated HG-mediated down-regulation of Bcl-2 expression (P<0.05).	salvianolic acid B	28-33	BCL2 associated X, apoptosis regulator	Rattus norvegicus	104-107	
32010242-7	2020	Western blot analysis determined that Sal B treatment significantly decreased the expression of pro-apoptosis proteins, including Bax, cleaved caspase-3 and -9, in spinal cord tissues after SCI but enhanced the expression of Bcl-2, an anti-apoptotic protein.	salvianolic acid B	38-43	BCL2 associated X, apoptosis regulator	Rattus norvegicus	130-133	
29257241-4	2018	Salvianolic acid B was identified to recover cognitive deficits and neurocytes, reduce inflammation, oxidative stress and neurocyte apoptosis (caspase-3 and Bax protein expression) in cerebral small vessel disease rats.	salvianolic acid B	0-18	BCL2 associated X, apoptosis regulator	Rattus norvegicus	157-160	
27614127-7	2016	Sal B effectively reduced vascular endothelial cell apoptosis, with Bcl-2 protein up-regulated and Bax protein down-regulated markedly.	salvianolic acid B	0-5	BCL2 associated X, apoptosis regulator	Rattus norvegicus	99-102	
28852731-7	2016	RESULTS: Sal B significantly improved heart function and decreased infarct size; remarkably decreased levels of serum TNF-alpha and IL-Iotabeta levels, increased contents of myocardium antioxidant enzymes activities; western blot results showed that Sal B ameliorate the increased Bax and caspase-3 protins expressions and decreased Bcl-2 proteins expression and ratios of Bcl-2 to Bax.	salvianolic acid B	9-14	BCL2 associated X, apoptosis regulator	Rattus norvegicus	382-385	
27139338-9	2016	Sal B inhibited the expression of Bax, cleaved caspase-9 and cleaved PARP, while promoted the expression of Bcl-2, LC3-II, Beclin1 and VEGF.	salvianolic acid B	0-5	BCL2 associated X, apoptosis regulator	Rattus norvegicus	34-37