PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32179247-0	2020	Salvianolic acid B promotes the osteogenic differentiation of human periodontal ligament cells through Wnt/beta-catenin signaling pathway.	salvianolic acid B	0-18	catenin beta 1	Homo sapiens	107-119	
32179247-11	2020	Sal B promoted the increase of ALP activity, osteogenic differentiation markers levels, mineralized nodules and activation of Wnt/beta-catenin signaling pathway, and DKK-1 could block those effects of Sal B on hPDLCs.	salvianolic acid B	0-5	catenin beta 1	Homo sapiens	130-142	
32179247-12	2020	CONCLUSION: Sal B promoted osteogenesis of hPDLCs through Wnt/beta-catenin signaling pathway, which providing a potential drug for periodontitis treatment.	salvianolic acid B	12-17	catenin beta 1	Homo sapiens	62-74	
28244648-6	2017	The passaged chondrocytes treated with Sal B showed strengthened cellular synthesis and stabilized mitochondrial membrane potential with upregulated expression of the marker genes for chondrocyte phenotype, Col2-alpha1, Acan and Sox9, the key Wnt signalling molecule beta-catenin and paracrine cytokine Cytl-1.	salvianolic acid B	39-44	catenin beta 1	Homo sapiens	267-279	
28391277-11	2017	Sal B-induced Wnt/beta-catenin pathway inactivation was blocked down by loss of lincRNA-p21.	salvianolic acid B	0-5	catenin beta 1	Homo sapiens	18-30	
28391277-15	2017	CONCLUSION: We demonstrate that lincRNA-p21-inhibited Wnt/beta-catenin pathway is involved in the effects of Sal B on HSC activation and lincRNA-p21 suppresses HSC activation, at least in part, via miR-17-5p-mediated-Wnt/beta-catenin pathway.	salvianolic acid B	109-114	catenin beta 1	Homo sapiens	58-70	
28391277-15	2017	CONCLUSION: We demonstrate that lincRNA-p21-inhibited Wnt/beta-catenin pathway is involved in the effects of Sal B on HSC activation and lincRNA-p21 suppresses HSC activation, at least in part, via miR-17-5p-mediated-Wnt/beta-catenin pathway.	salvianolic acid B	109-114	catenin beta 1	Homo sapiens	221-233	
17637199-4	2007	Immunoblotting results indicated that TNF-alpha exposure resulted in tyrosine phosphorylation of junctional proteins such as vascular endothelial cadherin and beta-catenin, which was attenuated by ESM and its active ingredients DSS and Sal B.	salvianolic acid B	236-241	catenin beta 1	Homo sapiens	159-171