PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34148127-14	2021	GSEA revealed that epithelial-mesenchymal transition, IL-6/JAK/STAT3 signaling, the inflammatory response, and TNF-alpha signaling via the NFkappaB pathway were remarkably suppressed pathways in patients with BRAF mutations.	gsea	0-4	interleukin 6	Homo sapiens	54-58	
35014680-8	2022	GSEA identified the IL-6/JAK/STAT3 pathway as a potential downstream signalling pathway of TGM2.	gsea	0-4	interleukin 6	Homo sapiens	20-24	
34712264-14	2021	Moreover, GSEA results indicated that the IL6/JAK/STAT3/SIGNALING pathway could be considered as a potential mechanism of genomic instability to influence tumor progression.	gsea	10-14	interleukin 6	Homo sapiens	42-45	
33891717-16	2021	GSEA showed that these genes are mainly related to "inflammatory response", "complement", "interferon-alpha response", "IL6/JAK/STAT3 signaling", "TGF-beta signaling", "IL2/STAT5 signaling" and "TNF-alpha signaling via NF-kappaB".	gsea	0-4	interleukin 6	Homo sapiens	120-123	
35205125-7	2022	Moreover, GSEA results showed that immune-related pathways, such as epithelial-mesenchymal transition and IL6/JAK/STAT3 signaling were enriched in the high ARPS risk groups, while the low ARPS risk group mainly regulated metabolism-related processes, such as adipogenesis and bile acid metabolism.	gsea	10-14	interleukin 6	Homo sapiens	106-109	
33790966-13	2021	GSEA showed that interleukin-6 (IL-6)/Janus kinase (JAK)/signal transducer and activator of transcription (STAT3) signaling, interferon gamma (IFN-gamma) response, angiogenesis, and coagulation were more highly enriched in the high-risk group and that oxidative phosphorylation was more highly enriched in the low-risk group.	gsea	0-4	interleukin 6	Homo sapiens	17-30	
33790966-13	2021	GSEA showed that interleukin-6 (IL-6)/Janus kinase (JAK)/signal transducer and activator of transcription (STAT3) signaling, interferon gamma (IFN-gamma) response, angiogenesis, and coagulation were more highly enriched in the high-risk group and that oxidative phosphorylation was more highly enriched in the low-risk group.	gsea	0-4	interleukin 6	Homo sapiens	32-36	
33194434-16	2020	GSEA analysis showed that several gene sets associated with tumor development and metastasis, including TGF-beta signaling, PI3K-AKT-mTOR signaling, and IL6-JAK-STAT3 signaling, were significantly enriched in POLR3G high expression phenotype.	gsea	0-4	interleukin 6	Homo sapiens	153-156	
33046027-10	2020	The GSEA results provided further functional annotations, including complement system, IL6/JAK/STAT3 signalling pathway and inflammatory response pathways.	gsea	4-8	interleukin 6	Homo sapiens	87-90	
32337262-8	2020	In addition, by GSEA, expression of CXCL1, CXCL11, CXCL2, and CXCL3 was correlated with several signaling pathways, including NOD-like receptor, oxidative phosphorylation, mTORC1, interferon-gamma response, and IL6/JAK/STAT3 pathways.	gsea	16-20	interleukin 6	Homo sapiens	211-214