PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33684397-8	2021	The binding mode of the drugs with M-protein was analyzed and it was observed that Colchicine, Remdesivir, Bafilomycin A1 from COVID-19 suggested drugs and Temozolomide from SuperDRUG2 database displayed desirable molecular interactions and higher binding affinity towards M-protein.	remdesivir	95-105	myomesin 2	Homo sapiens	35-44	
34305617-6	2021	In this study, we proposed that remdesivir strongly binds to membrane protein (Mprotein), RNA-dependent RNA polymerase (RDRP), and main protease (Mprotease) of SARS-CoV-2.	remdesivir	32-42	myomesin 2	Homo sapiens	79-87	
34305617-13	2021	Binding of remdesivir to Mprotease, Mprotein, and RDRP reduces the average motions in protein due to its strong binding.	remdesivir	11-21	myomesin 2	Homo sapiens	36-44	
34305617-14	2021	The MMPBSA calculations also suggested that remdesivir has strong binding affinity with Mprotein, Mprotease, and RDRP.	remdesivir	44-54	myomesin 2	Homo sapiens	88-96	
34305617-8	2021	It has been found that remdesivir binds to Mprotease, Mprotein, and RDRP with -7.8, -7.4, and -7.1 kcal/mol, respectively.	remdesivir	23-33	myomesin 2	Homo sapiens	54-62	
34305617-10	2021	It has been found that strong binding of remdesivir to Mprotein leads to decrease in structural deviations and gyrations.	remdesivir	41-51	myomesin 2	Homo sapiens	55-63	
34305617-12	2021	Furthermore, the eigenvalues and the trace of the covariance matrix were found to be low in case of Mprotease-remdesivir, Mprotein-remdesivir, and RDRP-remdesivir.	remdesivir	131-141	myomesin 2	Homo sapiens	122-130