PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26904147-8	2016	The possible mechanism might be through increased levels of leptin receptor and STAT3 and decreased PPARgamma expression, by which food intake of the rats was reduced and RSG-induced weight gain was inhibited.	Rosiglitazone	171-174	signal transducer and activator of transcription 3	Rattus norvegicus	80-85	
21599458-10	2011	Consistent with these effects, rosiglitazone treatment also decreased cleaved caspase-3 and p-STAT3 expression and, at the same time, increased SOCS3 expression.	Rosiglitazone	31-44	signal transducer and activator of transcription 3	Rattus norvegicus	94-99	
25662180-8	2015	The PPAR-gamma agonist, rosiglitazone, also inhibited leptin-induced STAT3 phosphorylation.	Rosiglitazone	24-37	signal transducer and activator of transcription 3	Rattus norvegicus	69-74	
21599458-11	2011	CONCLUSIONS: Rosiglitazone attenuated diabetes-induced apoptosis in retinal neurons through activities that may involve inhibition of p-STAT3 by induction of SOCS3, which suggested that rosiglitazone might be used to prevent retinal neuronal damage in diabetes mellitus.	Rosiglitazone	13-26	signal transducer and activator of transcription 3	Rattus norvegicus	136-141	
21599458-11	2011	CONCLUSIONS: Rosiglitazone attenuated diabetes-induced apoptosis in retinal neurons through activities that may involve inhibition of p-STAT3 by induction of SOCS3, which suggested that rosiglitazone might be used to prevent retinal neuronal damage in diabetes mellitus.	Rosiglitazone	186-199	signal transducer and activator of transcription 3	Rattus norvegicus	136-141	
17394460-9	2007	Rosiglitazone also increased the expression of the anti-inflammatory gene suppressor of cytokine signaling-3 and prevented the phosphorylation of the transcription factor signal transducer and activator of transcription-3 after focal ischemia.	Rosiglitazone	0-13	signal transducer and activator of transcription 3	Rattus norvegicus	171-221