PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10383397-2	1999	Previously we reported that 3-deazaadenosine (DZA), a potent inhibitor and substrate for S-adenosylhomocysteine hydrolase inhibits bacterial lipopolysaccharide-induced transcription of tumor necrosis factor-alpha and interleukin-1beta in mouse macrophage RAW 264.7 cells.	3-deazaadenosine	28-44	tumor necrosis factor	Mus musculus	185-212	
10383397-2	1999	Previously we reported that 3-deazaadenosine (DZA), a potent inhibitor and substrate for S-adenosylhomocysteine hydrolase inhibits bacterial lipopolysaccharide-induced transcription of tumor necrosis factor-alpha and interleukin-1beta in mouse macrophage RAW 264.7 cells.	3-deazaadenosine	46-49	tumor necrosis factor	Mus musculus	185-212	
9014821-2	1996	The 3-deazaadenosine analogues inhibited the TNF-alpha production and the inhibition was dependent upon the concentration of the analogue.	3-deazaadenosine	4-20	tumor necrosis factor	Mus musculus	45-54	
9014821-6	1996	The results show that the 3-deazaadenosine analogues are potent inhibitors of TNF-alpha production in the RAW264.7 cell line stimulated with LPS and suggest that these analogues may be effective agents for the treatment of diseases in which TNF-alpha plays an important pathogenic role.	3-deazaadenosine	26-42	tumor necrosis factor	Mus musculus	78-87	
9014821-6	1996	The results show that the 3-deazaadenosine analogues are potent inhibitors of TNF-alpha production in the RAW264.7 cell line stimulated with LPS and suggest that these analogues may be effective agents for the treatment of diseases in which TNF-alpha plays an important pathogenic role.	3-deazaadenosine	26-42	tumor necrosis factor	Mus musculus	241-250