PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31871450-1	2019	Aim: To clarify the contributions of fasting glucose (FG) and postprandial glucose (PG) to HbA1c in drug-naive patients with type 2 diabetes (T2D) and impaired glucose tolerate (IGT)/impaired fasting glucose (IFG).	pg	84-86	hemoglobin subunit alpha 1	Homo sapiens	91-95	
31871450-9	2019	Conclusions: In drug-naive patients with T2D or IGT/IFG, PG contributed more in patients with HbA1c < 8.5%, whereas FG became the predominant contributor in the poorly controlled patients with HbA1c >= 8.5%.	pg	57-59	hemoglobin subunit alpha 1	Homo sapiens	94-98	
18460560-11	2008	CONCLUSIONS: The main factors in support of using HbA1c as a screening and diagnostic test include: 1) HbA1c does not require patients to be fasting; 2) HbA1c reflects longer-term glycemia than does plasma glucose; 3) HbA1c laboratory methods are now well standardized and reliable; and 4) errors caused by nonglycemic factors affecting HbA1c such as hemoglobinopathies are infrequent and can be minimized by confirming the diagnosis of diabetes with a plasma glucose (PG)-specific test.	pg	469-471	hemoglobin subunit alpha 1	Homo sapiens	50-54	
32854906-8	2020	RESULTS: Elevation of PG was associated with elevated rate of LVEF<40%, STEMI, anterior wall location, DFG<60mL/min/m2 and higher troponin Ic pic (all P<0.001); HbA1c>5.9% was associated with elevated rate of CRP>3mg/L (P<0.001); high HbA1c and high PG together were associated with higher rate of MACE (P<0.001).	pg	22-24	hemoglobin subunit alpha 1	Homo sapiens	161-165