PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
14681026-7	2003	Moreover, FFSS induced Cx43 to migrate to the surface of the cell; this surface expressed Cx43 developed resistance to Triton-X-100 solublization.	Octoxynol	119-131	gap junction protein, alpha 3	Mus musculus	90-94	
17872368-5	2007	TNF-alpha did not affect total cell Cx43-PP2A catalytic subunit interaction, but it did induce PP2A catalytic subunit recruitment to the Triton X-100 insoluble subcellular fraction, in which Cx43-gap junction plaques are recovered.	Octoxynol	137-149	gap junction protein, alpha 3	Mus musculus	191-195	
17053190-8	2006	In accordance with the immunostaining results, immunoblots of the Triton X-100-insoluble fraction revealed an increase of Cx43 in gap junctions in extracts from Cx40-ablated atria, whereas total cellular Cx43 remained unchanged.	Octoxynol	66-78	gap junction protein, alpha 3	Mus musculus	122-126