PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22385953-3	2012	now identify two sphingolipids as specific cofactors for BAX/BAK activation that lower the threshold for apoptosis-associated cytochrome c release.	Sphingolipids	17-30	BCL2 antagonist/killer 1	Homo sapiens	61-64	
23750296-4	2013	We recently identified that two products within the sphingolipid pathway, sphingosine-1-PO4 and hexadecenal, directly regulate BAK and BAX activation, respectively.	Sphingolipids	52-64	BCL2 antagonist/killer 1	Homo sapiens	127-130	
22385953-4	2012	Association of mitochondria with other cellular membrane compartments is required for BAK/BAX exposure to these sphingolipids.	Sphingolipids	112-125	BCL2 antagonist/killer 1	Homo sapiens	86-89	
22385963-0	2012	Sphingolipid metabolism cooperates with BAK and BAX to promote the mitochondrial pathway of apoptosis.	Sphingolipids	0-12	BCL2 antagonist/killer 1	Homo sapiens	40-43	
22385963-3	2012	Biochemical purification of neutral sphingomyelinases that correlated with MOMP sensitization suggested that sphingolipid metabolism coordinates BAK/BAX activation.	Sphingolipids	109-121	BCL2 antagonist/killer 1	Homo sapiens	145-148	
22385963-6	2012	Furthermore, the sphingolipid products, sphingosine-1-PO(4) and hexadecenal, cooperated specifically with BAK and BAX, respectively.	Sphingolipids	17-29	BCL2 antagonist/killer 1	Homo sapiens	106-109