PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33050595-8	2020	ER stress induced by tunicamycin could clearly be reversed by activating LXRalpha because it promoted cholesterol efflux by enhancing the expression of ABCA1 and ABCG1 in hepatocytes and macrophages, contributing to attenuation of the development of AS.	Tunicamycin	21-32	ATP binding cassette subfamily A member 1	Homo sapiens	152-157	
22497927-8	2012	Tunicamycin, a classical ER stress inductor, also impaired ABCA-1 expression and cholesterol efflux (showing a decrease of 61% and 82%, respectively), confirming the deleterious effect of ER stress in macrophage cholesterol accumulation.	Tunicamycin	0-11	ATP binding cassette subfamily A member 1	Homo sapiens	59-65